COPD and Emphysema Self-Care (Part 1)

An emphasis on Chest Physiotherapy and Breathing Training

This Knol is directed mainly at patients with symptomatic COPD and Emphysema, though persistent severe Asthma might also benefit. The emphasis is on patient self-care and empowerment, mainly in the home setting. Those in Pulmonary Rehabilitation programs might find some of these dyspnea relief breathing techniques of interest.


Introduction

This is Part 1 of a four part series of Knols on this subject.

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COPD and  EMPHYSEMA   SELF  CARE
A Manual For Patients And Their Families
© 1987, 2005, 2006, 2008  Deane Hillsman, MD

I am Dr. Deane Hillsman, a retired physician who practiced Internal Medicine and the specialty of Pulmonary Diseases. My particular interest is Pulmonary Rehabilitation, and chest physiotherapy and breathing training to help people with COPD and related problems.

In 1987 I wrote the draft of a book RESPIRATORY SELF CARE: A Manual For Patients And Their Families. It was about 60% completed, and I showed it to a half-dozen or so of the major publishers of this type of work. None of them were interested in taking it on. Their concerns were generally that, the material was too technical for most patients and their families, and I therefore abandoned the project.

Times have changed, and in recent years patients and families are now demanding more in-depth understanding of their health concerns, and the concept of patient self-help and empowerment is fast becoming a significant factor in health care. I reject the notion that patients are somehow not capable of using more advanced health care information to their advantage. The contents of this web site are substantially taken from that long abandoned manual.

In the early 1960's during my residency training, quite by accident, I was privileged to see two British trained Physical Therapists work with a patient suffering from Emphysema using  breathing exercises  (which I had never heard of), and achieve significant dyspnea (shortness of breath) relief. I was impressed.

I was later told by many that  breathing exercises  were not of value in COPD. However, when you see such positive results it is difficult to ignore what you have seen. This led to a lifelong interest in trying to understand, and improve, the technology that is generally known as Chest Physical Therapy (or Chest Physiotherapy or simply  Chest Physio ). The modern name for those credentialed in this specialty by the American Association of Physical Therapy is Cardiovascular and Pulmonary Physical Therapist.

A major part of Chest Physiotherapy is  breathing pattern  training. This relates to how to breathe, to achieve more efficient patterns of breathing. To further this interest, in the 1970's I invented a sophisticated computer based device to visually define breathing pattern templates, and to visualize patient's real-time breathing, and their interaction with different breathing templates to achieve maximal breathing comfort. In other modules in this series you will see patient examples from that system. The Breathing Trainer, to be described later, is a simpler version of that original professional and research system, specifically designed for individual home use.

It is quite astonishing, but true, the scientific pulmonary community has never defined, even to this day, in any comprehensive manner, the most efficient way for a COPD / Emphysema patient to breathe. Because of this lack of scientific guidance as to how to program my breathing training system, I had to rely on the general guidelines provided by the Chest Physiotherapy community. And later, I observed a few patients in my practice with very advanced Emphysema, but with only mild complaints of dyspnea. I collected their breathing patterns, and observed they correlated well with the descriptions of Chest Physiotherapy techniques. Indeed, these few patients appeared to be a model of natural adaptation to the altered breathing mechanics of COPD. The lessons learned from these few very instructive patients have substantially guided my design of breathing pattern templates for others with COPD.

The first prototype of my breathing training invention was presented in 1978 before the California Thoracic Society. Since then I have had a great deal of learning experience with experimental adjustment of the various parameters that go into characterizing a breathing pattern for individual patients. Essentially I was trying to understand the optimal manner for the COPD patient to breathe comfortably. I am impressed how even small adjustments can make a significant difference in breathing comfort to many of these patients.

Chest Physiotherapy is substantially a creation of British Physical Therapists, and the subject is routinely taught in the U.K. schools of Physical Therapy. Chest Physiotherapy began there in the mid-1920's and was quite mature by the mid to late 1930's.

This ancient 1935 training manual obtained from the British Library is a quite remarkable document.


It is clear from this document, at a time when the physiologic understanding of COPD clinical problems was primitive, these early Physical Therapists understood the importance of chest overinflation (more later on this very important topic of Dynamic Hyperinflation), and basically how to correct this functional lesion.

How these early Chest Physiotherapists managed to figure this one out is remarkable, and certainly worthy of admiration.

Also quite astonishing is the general lack of awareness of this technology in North America. Surveys of general Physical Therapy training programs have documented usually only minimal time devoted to the subject. In fact, today there are only about 130 credentialed Cardio-Pulmonary Physical Therapy specialists in the United States. If COPD patients seek Chest Physiotherapy training, they are not likely to be able to find these services, at least from these properly qualified experts.

There is also ongoing and substantial physician resistance to the concept of breathing training, as  breathing exercises  are in some disrepute. Perhaps this is related to the many non-physicians offering various breathing services and devices, for many reasons, some of them of dubious value. Physicians instead have understandably relied on the very complex traditional regulatory feedback mechanisms that are essential to maintain life.

Are these traditional breathing control mechanisms infallible? Until recently most physicians appear to have had total faith in these important traditional control mechanisms. However, as we will see, that apparent traditional faith has not been justified. As we shall see, these natural breathing control mechanisms can indeed be tricked into abnormal pathologic behavior, to cause increased shortness of breath.

Recent research has characterized the breathing pattern response to exercising COPD patients, and patients who breathe too rapidly, as a  vicious circle  response, leading to the physiologic problem of progressive so-called  Dynamic Hyperinflation  of the lungs, and this in turn causing severe dyspnea limitation. Simply stated, this rapid breathing pattern causes too much air to become trapped in the lungs, and the lungs can no longer work properly.

This is a pulmonary mechanical defect, and any feedback control system that has a vicious circle mechanism causing failure of the overall system is a flawed system. An obvious option for the COPD patient seeking breathing comfort from a mechanical defect is a proper mechanical breathing control technique, in addition to the usual bronchial dilator drugs and other medications typically prescribed for shortness of breath. Corrective breathing mechanics is what breathing training is all about, and we will discuss this important topic of Dynamic Hyperinflation in a later instruction module.

This breathing training debate should now really be over, though many pulmonary physicians might still dispute that statement. The option of breathing control for patients with COPD and Emphysema, and for Asthma exacerbations, is indeed an important, and in fact necessary option for optimal breathing comfort. By definition, bronchodilator medications can never completely correct the airway obstructive problem in COPD, and in those patients with moderately severe or severe disease, the majority of the mechanical airway obstructive lesion persists after bronchodilator therapy. This residual mechanical problem requires a mechanical solution, and that is what breathing training strategies are all about.

The majority of formal pulmonary rehabilitation programs have a primary exercise focus, or an exercise-centric focus. These programs emphasize reconditioning the legs and other peripheral muscles, by using stationary bicycle ergometers, or treadmills, or stair climbing. Exercise makes these muscles more efficient as to oxygen needs and getting rid of waste carbon dioxide. This requires relatively less breathing, and therefore less dyspnea, to satisfy these metabolic oxygen and carbon dioxide requirements. Considerable research has been done in this approach, and there is no question that this is an effective strategy to get patients ambulating, and to reduce their overall level of dyspnea. And as patients feel more comfortable, they are able to get about more easily. However, exercise training that makes one more short of breath is not a pleasant training experience.

The other general approach, and the one that I personally favor, is a primary breathing training strategy, or a breathing training-centric focus. Ask COPD patients what their major concern is, and very few will complain about the inability to engage in exercise. COPD patients dominant concern is mainly about general and acute shortness of breath, and the large majority are most concerned about acute dyspnea attacks. Acute dyspnea is a very distressing experience, and COPD patients live in constant fear of these attacks. It makes logical sense therefore that initial therapy should concentrate on the patient's primary complaint of dyspnea.

Remember Willie Sutton, the famous bank robber? As Willie Sutton allegedly said, when asked why he robbed banks,"  .....Because that's where the money is....."     Applying this analogy, think of the primary breathing control focus in COPD rehabilitation as the Sutton approach to pulmonary rehabilitation, because that indeed is where the action is.

Teach COPD patients breathing control and their overall breathing comfort improves. But more important, if patients know how to recognize an impending acute breathing attack and prevent the attack, or if having an attack how to control the attack, they will lose their fear of exerting themselves. And if the patient feels they are in control of their breathing, it takes only a little encouragement to get them to be more active, because patients usually want to be more active. And with increased activity, their leg and other peripheral muscles will become reconditioned, and strength will improve, and their overall rehabilitation program will therefore be enhanced. If you will, this is an alternative option to provide exercise reconditioning.

Please do not think that the techniques that I will be showing you are a complete substitute for proper instruction from a qualified therapist. However, if you do not have access to a qualified therapist, the modules of instruction I am providing may be of use to you.

And if you are presently receiving instruction in  "breathing exercises"  or  "diaphragmatic breathing"  perhaps your therapist might be interested in a few of the techniques and tips in these instruction modules.

It is my hope this series will provide information to patients, and their families and caregivers, that will empower them, in a self-help manner, to use these lessons to gain dyspnea control and relief in their activities of daily living. And when you have mastered dyspnea control, that you will then be able to be even more active, and more comfortable.

Throughout these instruction modules, which will require a lot of work on your part, please always remember, neither I nor your doctor or therapist can do dyspnea control for you. We can instruct you, but only you can do dyspnea control for your needs.

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The First Step

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At the end of the consultation with a new COPD / Emphysema patient I usually told them:
 I can't help you, but I can show you how to help yourself.

Yes, this was rather harsh, but it did get their attention. And No, nobody got up and walked out on me, though I did wonder about this possibility a few times.

This deliberate strategy was a realistic technique to hopefully set the stage for the pulmonary rehabilitation program to follow. Patients are usually oriented to the belief that their doctor is simply going to provide them with some pills, and that by taking these medications their health problems will be resolved. True, taking medications in many disease conditions will resolve their problems. However, for the COPD / Emphysema patient nothing could be further from the truth. Effective therapy for these patients is critically dependent on patient understanding, cooperation, and various respiratory skills. It requires the patient to become a part of a team effort, with their doctor and therapists.

I then told them the first crucial lesson to be learned is:
 Every breath of air first begins by getting the old stale air out, to make room for the fresh air.

This is a very counter-intuitive message to patients, as their natural focus is to get air IN, to relieve their dyspnea. To persuade patients as to the importance of properly exhaling their last breath, it is helpful to describe the residual last breath in derogatory terms such as  dead air  or  foul air  or  old bad air  and similar terms. The object is to change the patients focus from inspiration to expiration. Indeed, and as you will learn in greater detail in lessons to come, the expiration phase of breathing is the most crucial and difficult to learn.

And finally, I gave them some simple instructions for a much abbreviated breathing control program. Most of the time patients obtained at least some immediate measure of dyspnea relief, and if they did it was an excellent beginning. I could then advise them this was a favorable sign for better things to come, when they had developed more advanced breathing control skills. The instructions are as follows:

 Sit back in a comfortable easy chair (or if in bed, propped up on at least three pillows). Relax, you can't breathe properly if you are tense and anxious. Breathe gently, and rhythmically. Slow down your breathing. Concentrate on breathing OUT, and MAKE YOUR EXPIRATIONS LONGER. When you breathe in, take in a gentle, slightly larger breath, and try to place, and try to feel the air going down to your lowest lateral ribs, directly in line with the anterior portion of your arm pits.

A good trick to get the very important proper chest movement is as follows. Raise your elbows up, with fingers hanging down. Now, curl your fingers backward, so the back of your fingers are facing the floor. Then, place the back of your fingers on the very lowest ribs (next to your abdomen), directly under your arm pits.

As you breathe in, try to direct your breath toward your fingers, and feel this part of your lower chest moving out and sideways. Try to imagine putting all of your breath into this part of your chest.

Try it. It may not work the first few times, but keep trying. A friend or spouse coaching you with these instructions, and putting their hands on your lower chest in the manner as described above, may help to get you started.

And if you do have some success with these primitive instructions, this is indeed a sign of better things to come, when your chest is mobilized and working better, and you have mastered some breathing skills.

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Understanding Breathing Attacks

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There are several reasons why you might be having a  Breathing Attack  or  Acute Dyspnea Attack.  Essentially this is an exacerbation of dyspnea (shortness of breath) on top of whatever background of chronic dyspnea you might have. The five commonest reasons for these dyspnea attacks are:

    *  Exertion
    *  Coughing up mucus
    *  Bronchospasm ( Asthma ) exacerbation
    *  The Rescue Breathing Pattern
    *  Dynamic Hyperinflation

1.) EXERTION
This is the commonest cause of a dyspnea attack. You have exerted yourself beyond the point where your lungs can provide the necessary increased ventilation to meet your increased metabolic needs (i.e. taking in more Oxygen and getting rid of more Carbon Dioxide) due to increased activity. A crucial skill you must learn is measured pacing of your activities at a slower and lower effort level, for whatever particular activity you are doing. We will discuss this in detail in another module, as well as some breathing strategies to help this problem.

2.) COUGHING UP MUCUS
Coughing is normal and necessary to clear mucus ( phlegm ) from your lungs. However, should you cough up a larger blob of mucus from deep within the lung, and it sticks in the larger airways, this can precipitate a more violent coughing and choking spell. This is particularly common in the hour or two after waking up, as mucus has been accumulating overnight, and tends to be thicker and stickier. We will discuss this in detail in another module, and show you the more efficient Huff Cough technique used in COPD.

3.) BRONCHOSPASM ( ASTHMA ) EXACERBATION
Bronchospasm refers to a spasm contraction of the muscles in the bronchial tubes, thereby making them narrower and therefore more restrictive as to being able to move air freely. It is commonly called an  Asthma Attack,  but technically Asthma is a separate entity from COPD, though indeed there is some overlap of the two conditions. It is better to use the term  bronchospasm  if you have COPD, because directions for treating true Asthma that you might hear of, may, or may not, be appropriate for COPD. We will discuss this in detail in another module, and show you the Metered Dose Inhaler (MDI) technique to inhale so-called  Rescue Medications  into your lungs for faster and more effective asthmatic or bronchospasm relief.

4.) The RESCUE BREATHING PATTERN
This is a very common breathing pattern of rapid and more forceful breathing, which develops when patients get upset or panicky. The basis for this problem is entirely psychological, but the consequences have serious physiological implications. It is often seen when patients develop some shortness of breath for whatever reason, and then become (understandably) upset, acutely precipitating this abnormal breathing pattern. This is most unfortunate, because they will then develop further dyspnea. Rapid and forced breathing is very detrimental to COPD breathing control. We will discuss this in detail in another module, and show you techniques to control this problem.

5.) DYNAMIC HYPERINFLATION
Dynamic Hyperinflation refers to overinflation of the lung, because the air you have inhaled does not have sufficient time to fully exhale, and therefore your lungs progressively inflate into a position where breathing becomes much more difficult.

Dynamic Hyperinflation is commonly seen with acute Asthma attacks, as well as COPD bronchospasm attacks. And as noted above, in COPD patients exerting themselves and breathing faster. Dynamic Hyperinflation prevention and/or correction is a major reason why breathing control techniques work in COPD. It is therefore crucial you understand this concept. We will discuss this in detail in another module, and show you techniques to control this critically important problem.

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Breathing  Control  Overview

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Note: In order to easily follow the description of this diagram, it is suggested that you first print it.
You can do this by right clicking on the diagram, and then use the commands "View as" or "Copy" (or similar words). This will put the diagram in the computer clipboard, and you can print it from there.

Breathing training with the Breathing Trainer for COPD needs to be put in perspective, and the following diagram briefly summarizes some of the major physiologic components.


Breathing training and the Breathing Trainer is only part of the equation in breathing control, though certainly a very important part. It can show you very effectively how to breathe, but no breathing is possible without an effective chest and diaphragm so-called "Bellows" mechanism to make the lungs actually move. All active lung movement is totally dependent on an effective Bellows mechanism to enable more efficient breathing pattern training. It is therefore important you have some understanding of the bellows mechanism. In another module we will describe how the COPD chest becomes deformed into the so-called "Barrel Chest" deformity, the physiologic consequences of this, and how to deal with that problem with chest physiotherapy techniques.

The Bellows mechanism is composed of the "Chest Wall" as noted in the center of the diagram. The Chest Wall in turn has two distinct components, the "Ribs" of the chest, and the "Diaphragm," which is a thin curved muscle between the chest and the abdomen, attached to the lower ribs. The diaphragm is the major driving force of breathing, and in COPD it's function is typically severely compromised, by lung overinflation that pushes the diaphragm downwards into the abdomen, putting it into a position of mechanical inefficiency. It is critically important that diaphragm function be restored as much as possible, in order that you can then effectively use breathing training to learn more efficient breathing patterns.

The breathing pattern parameters are defined in the center of the diagram, by adjusting:

    * Tidal Volume (i.e. the breath volume of air)
    * Respiratory Rate (i.e. the number of breaths per minute)
    * Inspiration : Expiration Time Ratio (i.e. the relative time of the Inspiration and Expiration components)
    * End-Inspiration and End-Expiration Pauses (i.e. slight breath hold times at the end of inspiration and expiration)
    * Inspiration and Expiration Waveforms (i.e. the shape of the inspiration and expiration breathing pattern. NOTE: The complexity of waveform considerations has been omitted from the Breathing Trainer.)

As noted in the small diagram on the right, it is critical the breathing pattern achieve a minimal adequate degree of "Alveolar Ventilation," i.e. the ventilation breath that actually gets down to lung alveoli (air sacks) where gas exchange takes place.

However, as noted in the small diagram on the left, it is also critical that the breathing pattern produce the minimal degree of "Work of Breathing" i.e. a measure of the effort and energy to breathe, as dyspnea is most closely related to the increased Work of Breathing.

It should be apparent that there are conflicting needs involved in developing an optimal breathing pattern, and that balancing these different breathing parameters of adequate Alveolar Ventilation versus minimal Work of Breathing is a delicate task, and one that requires compromises.

Considerable experimental adjustment may be needed to achieve the optimal compromise. I am impressed as to how small adjustments may make substantial differences in patient comfort. The Breathing Trainer is designed to permit very subtle adjustments to permit you to seek out the breathing pattern that is best for you. Another module will go into detail as to how to adjust your Breathing Trainer to make a "Breathing Prescription" individualized for your particular needs.

Be aware there are many therapists who strongly advocate for one or another type of breathing pattern. I would suggest that frequently a strong advocacy position does not take into account the fact that most every breathing parameter adjustment has both positive benefits, and also undesirable negative factors. The trick is to find the optimal balance between these conflicting parameters.

Please do not skip directly to the module on Breathing Trainer adjustment. It is suggested you proceed to the Barrel Chest module, to better understand the underlying problems that must be corrected by chest physiotherapy.

The more you know about these conflicting parameter requirements, the better the results you will achieve in your rehabilitation program.

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The  Barrel  Chest  Deformity

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COPD / Emphysema classically produces the "Barrel Chest Deformity" as noted in this diagram from Dr. Frank Netter.

These changes develop slowly, over many months and years. As such, the early Barrel Chest may be subtle and difficult to recognize, but it is still of importance.

Note the chest is generally overinflated. This is because the lungs are overinflated, and pushing the chest wall out.

Also note the humped back deformity, called Kyphosis, pushing the upper chest and neck forward. Poor general posture is a significant result, and it impacts unfavorably on your ability to breathe.

When pushed out in this manner the normal outward rib movement of the lower / lateral chest becomes limited. And with limited movement comes stiffness, and further movement limitation. This movement limitation will limit your ability to take in a deep breath.

As a result the chest wall becomes "frozen," and the entire chest now is less capable of expanding and therefore further limiting larger breaths. Also, the "frozen" state now results in the chest now moving as a single unit, so-called "Unit Movement," instead of the two distinct chest movements as noted below.

The result is a shift to inefficient abnormal upper chest breathing, using the so-called "Accessory Muscles" of breathing, and therefore a decrease in the normally dominant outward lower / lateral chest breathing. The diaphragm is attached to the lower ribs, and normally has a coordinated and synergistic movement with these ribs. Without this coordinated movement, diaphragm function is significantly impaired. It is therefore very important to restore proper outward lower chest movement in order to allow better diaphragm function. And likewise to minimize the upper chest movement, which should only be used in emergency breathing situations.

Remember, the diaphragm is the major muscular organ that drives breathing, and therefore restoring that function is critically important.

To give you a better understanding of the two distinct chest movements, consider the anatomy of the ribs, as taken from Grant's Atlas of Anatomy.


Note the upper ribs are short and relatively straight. The muscles that move these ribs are centrally placed, and therefore pull the chest directly upward and outward, with the so-called "Pump Handle" movement. With the "frozen" chest, this is what pulls the chest upward and outward with the so-called "Unit Movement."

The lower chest movement is more complex. Other than for the almost straight "short ribs" 12 and 11, the lower ribs are sharply curved. Between these ribs are the "External Intercostal Muscles" which that slant forward and downward to the ribs below. When these muscles contract, the ribs are pulled in an upward and lateral direction, which expands the lower chest. This is the so-called "Bucket Handle" movement.

This outward and lateral Bucket Handle movement is what stretches the attached diaphragm muscle into a more favorable position of movement, and what restores some of the important synergistic movement between ribs and diaphragm.

This diagram taken from Cherniack and Cherniack's text "Respiration in Health and Disease" brilliantly illustrates the complex rib movements of the "Pump Handle" and Bucket Handle" movements.


Note the different actions between the 3rd and 9th ribs.

As the large majority of lung volume is in the lower chest, and considering this illustration of the Bucket Handle movement, can there be any doubt as to the importance of lower / lateral / outward chest movement in restoring diaphragm function?

We will now move on to the topic of chest mobilization, chest movement training and coordination, and chest strengthening, using Chest Physiotherapy techniques. And then the all important topic of Dynamic Hyperinflation, and using breathing strategies to prevent or correct this problem.

With the knowledge you have from these preliminary modules you will then have better understanding of the underlying basics, and why we are asking you to practice certain techniques. And you will be in a better position to learn about effective breathing pattern training.

In other modules we will elaborate on general posture improvement, including neck positioning, as well as some specific posture tricks that will assist your breathing.

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Chest  Physical  Therapy


Mobilizing, Training and Strengthening the Chest

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Your chest must first be mobilized because, as you have learned, it is out of correct positioning and stiffened. Next it must have corrected and coordinated movement, because it is not moving properly. And finally the chest muscles must be strengthened, because the immobilized chest has permitted weakness to develop in your breathing muscles .

This is a complex task, and ideally would require a skilled professional Physical Therapist (Chest Physiotherapist) to use "hands on" techniques to augment chest movements and teach corrective and coordinated movements, and enhance joint flexibility with local massage and supplemental heat or diathermy. However, as previously indicated, these services are difficult to find in North America, as indeed there are presently only about 130 credentialed Cardio-Pulmonary Physical Therapists in the United States. We will provide you with self-help instructions that will cover the major points of this technology, but regardless of these instructions, you are encouraged to use them in conjunction with your physician and appropriate other health care providers.

However, there is another option you might consider, and that is Yoga. Dr. Vijai Sharma, a practicing clinical psychologist and credentialed Yoga instructor with a particular interest in COPD, has developed training DVD videos designed specifically for home use by people with COPD. Dr. Sharma is currently offering two DVDs and instruction manuals:
"Stretching Breathing Exercises adapted for people with severe COPD" and
"Stretching Breathing for COPD For all levels of fitness".

The various Yoga spine and neck exercises, coupled with Yoga breathing exercises and breathing coordination with general body movements, seem well suited to substituting and/or complementing traditional Chest Physical Therapy, and some aspects of traditional Occupational Therapy. Dr. Sharma has a web site with many helpful COPD instructions, and in particular as to COPD anxiety and depression. His instruction videos may be purchased at his web site (  www.mindpub.com ).

Physical Therapy is much involved with so-called "Adaptive Substitution Movements or commenly-called "Trick Movements," which basically is the use of different muscular groups to assist the function of impaired neuromuscular groups. There are "Good Trick Movements" (i.e. Adaptive Substitution Movements) which are productive, and "Bad Trick Movements" (i.e. Mal-Adaptive Substitution Movements) which are not efficient or productive.

Bad Trick Movements may develop during the course of a disease, and they may also appear any time one is doing therapeutic neuromuscular training. It is therefore important to watch out for bad trick movements developing, and to correct them. In diaphragmatic breathing training there is one common bad trick movement called the "Belly Puffing Artifact" that must not be allowed to happen.

The  BELLY  PUFFING   ARTIFACT

The Video for this is in the Chest Physical Therapy module

 Normally when one inhales the diaphragm descends toward the abdomen, and as a result the abdomen rises. However, it is important the abdomen rise naturally because of proper rib and diaphragm movement. Belly Puffing, which is not due to diaphragm movement, can mimic normal abdominal protrusion due to correct diaphragm movement. And unfortunately, Belly Puffing can be easily learned. Try this exercise in Belly Puffing on yourself, to see how easy it is to do, and so you can recognize it during your diaphragmatic breathing training.

While standing, breathe IN deeply, and at the same time suck your abdomen IN. Then exhale fully, and while doing so, puff your abdomen OUT. Note this is 180 degrees out of phase with the normal abdominal movement due to diaphragm action. Now, lying flat on your back, do the same Belly Puffing maneuver, and note how easy it is to do. Belly Puffing is not simply a shifting of thoracic-abdominal contents by gravity.

Abdominal Belly Puffing that is 180 degrees out of phase with true diaphragm action is called "Paradoxical Chest Movement," and it is usually easy to detect. However, much more common, and much harder to detect, are partial forms of Belly Puffing, producing various degrees of "Asynchronous Breathing" that may be easily confused for true diaphragm movement. With more severe degrees of Asynchronous Breathing you may think you are getting good diaphragmatic breathing, but in fact your diaphragm movement may be substantially sub-optimal.

The only true abdominal indication of diaphragmatic breathing is the rise of the abdomen that flows naturally from the action of the rib Bucket Handle movement, and this doesn't happen until about 1/4 to 1/3 or more into inspiration. Abdominal puffing prior to this inspiration timing is Belly Puffing. Even more subtle and difficult to detect, is partial Belly Puffing later during inspiration.

There are two commonly practiced diaphragmatic breathing techniques that should not be done, because they have a substantial chance they are in fact teaching Belly Puffing, not true diaphragmatic breathing training.



Note one hand placed on the upper chest, and the other on the abdomen, just below the ribs. Typically this exercise is done from the sitting or lying position.

The patient is instructed to not move the upper chest while breathing in (i.e. to correct the abnormal "Pump Handle" movement), and at the same time to have the abdomen come out with inhalation, to teach diaphragm movement.

Note however, these instructions are in fact likely to teach Belly Puffing, not true diaphragm breathing.

The proper instruction is to have the patient direct their inspiration breathing to the lowest rib margin, directly beneath the anterior margin of the arm pit. This will make the chest move outwards and upwards because of the "Bucket Handle" movement. There should be no abdominal movement for about 1/4 to 1/3 of inspiration, and at that point the abdomen will then begin to protrude outwards. But this time abdominal protrusion is the result of true diaphragm movement.

All abdominal movement on inspiration should flow naturally from correct "Bucket Handle" chest movement.

Another problematic technique which is commonly taught, is, while lying down, to have a weight just below the ribs, and to concentrate on making the weight rise on inspiration. This is similar to the two hand technique noted above.

By focusing on making the weight rise, again, this has obvious potential to teach Belly Puffing.

However, this technique can be a useful one, provided the therapist uses two flat sand-bag type weights, each positioned along the lower-lateral rib margin, with no more than half the weight lying on the abdomen. If the patient watches the weights move in this position, they will now have a visual biofeedback prompt to encourage performance of the Bucket Handle chest movement. In addition, with graded weights this exercise may enhance chest wall strength.

The correct hand position for diaphragm breathing training is as noted.
Note the hands on the lower rib margins, directly below the anterior portion of the arm pits. The fingers, preferably only the distal half of the fingers, are over the lowest ribs and on the abdomen. The inspiration breath should be gently directed towards the palms of the hands, which should rise gently. And try to feel the air moving into this region. Learn to recognize this feeling of correct breathing.

There is an alternate hand placement trick, to guide you to the correct Bucket Handle movement, which some patients find easier to do. Raise the elbows up to shoulder height, and let the hands dangle. Then, curl the fingers until they are facing the floor. Then place the backs of the fingers on the very lowest rib margin, directly under the arm pits.

As the ribs swing outward and upwards, with a little practice about 1/3 of the way through inspiration you should be able to feel the diaphragm gently rising against your finger tips. And about this time the abdomen should gently rise, but this time because of true diaphragm movement. The hand positioning is providing the signal to train you in correct diaphragm breathing. With only a month or so of diligent practice you should be able to do this type of breathing naturally, and without using the hand prompting signal.

Note that I have made no mention of inhibiting the abnormal upper-anterior "Pump Handle" chest movement, which commonly taught, and which causes the upward "Unit Movement" of the chest. This is seldom needed. If indeed you correctly focus on the correct "Bucket Handle" movement this upper chest movement will gradually disappear. Occasionally however, some directed voluntary suppression of this upper chest movement is needed.

Generally you should teach yourself to recognize this abnormal upper chest movement, and during quiet breathing, to voluntarily suppress this upper chest movement.

However, If your breathing reserves are low, and at times of breathing distress, you may need to use your accessory muscles of respiration to employ the Pump Handle movement as an emergency breathing mechanism. This is an advanced breathing technique. The important point to remember, is to let any upper chest movement flow naturally from the dominant lower chest Bucket Handle movement. The Bucket Handle movement is always primary, and the key to success. Just as abdominal diaphragmatic movement flows later from the Bucket Handle movement, so to does the upper chest Pump Handle movement flow later from the Bucket Handle rib action.

You will need this upper chest suppression skill to help correct the Rescue Breathing Pattern. More on this subject in a later module.

BREATHING  BELT  EXERCISES


The Video for this is in the Chest Physical Therapy module

The Breathing Belt is a simple device used by Chest Physiotherapists to apply directed pressure to the ribs for chest mobilization and breathing enhancement. And as you will see, it has other useful purposes. But first you must know how to construct a Breathing Belt.

How To Make a Breathing Belt:

Take an old sheet and cut a piece 14 to 16 inches wide down the entire length. The eventual length will be approximately from floor to shoulder height, but you can correct that length later. Place the two edges together lengthwise, and sew them along the edge, to make about an 8 inch wide piece. Then fold the edges together again, and sew them along the edge to make about a 4 inch wide belt. You can make the belt a little fancier by inverting the entire piece after sewing the edges. To keep it from getting distorted during use, run two or three evenly spaced stitches down the middle. And a zigzag or wavy crosswise stitch will further prevent the material from distortion during use.

The Safety Belt


The Breathing Belt may also be used as a Safety Belt. This is an old technique used by Physical Therapists to help ambulate frail patients safely.

Tie the belt snugly around the lower waist of the patient, just above the brim of the pelvic bones, and secure it with a knot at the back (no safety pins or insecure clasps). Then, the person assisting the patient should firmly grasp the belt with one hand in the mid portion of the back. The other hand may be used to otherwise assist the patient.

The Safety Belt may then be used to assist the patient in getting out of bed, getting up from a chair, or walking. Always maintain the belt hand, as this is the controlling hand should the patient lose their balance, or look in danger of falling due to weakness.

Most of the falling accidents happen because a frail patient loses their balance. However, with the controlling hand on the Safety Belt even a small helper can easily control most patient acts of incoordination and stumbling.

And should the patient actually fall, the controlling hand on the Breathing Belt can ease the patient to the floor without serious injury.

Positioning the Breathing Belt


The Breathing Belt is best used in the sitting position, though it could be used both standing and lying down.

Place the belt behind you, at the level of the lowest ribs. The lowest portion of the belt should be positioned about an inch below the anterior lowest rib, directly below the collar-bone (or Clavicle). It is important the belt not be lower than this, or you will only be compressing the abdomen, and therefore deriving no rib mobility benefit.

Now, take your right hand and grasp the left belt, just below the anterior portion of the arm pit. And cross your left hand over to the right belt at this same position below the arm pit. Many patients find it convenient to grasp the belt as though one is holding the reins of a horse.

You are now ready to do chest mobilization.

Relax. As with all breathing techniques it is important that you do them in a relaxed manner.
Take in a slow, deep and gentle breath, and then totally relax your chest and let the air gently fall out of your chest.
Then, about half way through breathing out, apply chest pressure by pulling your hands towards each other, directly across your chest. As you are pulling, increase the pressure gradually and firmly. Do not pull suddenly or forcefully, as this type of pressure could crack or even fracture a frail rib. Try to get the sensation as though you are wringing water out of a wet bath towel. Make your exhalation time prolonged, at least two to three times your normal time of breathing out.

Then, simultaneously, release the belt pressure and breathe in gently and fully. Direct this inspiration breath down to the bottom of your lungs, and laterally, directly below the anterior portion of your arm pits. This is critically important, as this action is training your rib muscles to do the Bucket Handle movement. Try to feel the air moving into these lower regions of your lungs.

If you have done this correctly, release of the belt pressure should cause your compressed ribs to spring out, and you should feel a satisfying rush of air into your lungs. Repeat this compression cycle, and try to get a rhythm to your chest compressions.

Patients frequently get confused as to when to apply the belt pressure, as indeed the combination of breathing phase and belt pressure is somewhat counter intuitive. If you get these movements mixed-up, the application of the breathing belt pressure will work against your breathing, and your breathing then will immediately get worse.

Remember:
Pull and Squeeze to breathe out;
Relax and let go to breathe in.

Done with skill, and without excessive compression force, this can be a useful trick to relieve an acute attack of dyspnea which has resulted in Dynamic Hyperinflation.

For rib mobilization exercises, three to five minutes, done twice or at most three times a day, should be sufficient. Done more than this the exercises can become unpleasant and boring, and you may lose interest. But during these brief practice times you should concentrate on technique perfection, and particularly where to place your inspired breath of air down in the lower-lateral lungs.

A warning: Stiffened ribs that are being mobilized often complain by producing a general aching type of pain, and this discomfort usually takes about five to six weeks to slowly resolve. Local low-level gentle heat and simple pain relievers such as Aspirin may help. However, a sharp localized pain may indicate a cracked or broken rib, and you should stop further belt exercises until you have been checked by your doctor. Be patient. You will find that this discomfort is well worth the trouble if you regain the capacity to take in deeper breaths, and being able to take in deeper breaths easily is what this is all about.

CHEST  WALL  COORDINATION


Muscular movement is seldom as a result of a single muscle moving in one direction. Muscles act together in groups, that support the activity of one another in a coordinated and synergistic manner. Much of that coordination and synergism of breathing has been lost in the development of the "Barrel Chest" deformity of COPD.

As noted previously, much of the abnormal COPD chest movement is the stiff upward "Unit Movement" involving the "Pump Handle" action. And because of the overinflated positioning and chest stiffening of the lower chest, the crucial "Bucket Handle" movement is now minimal, which results in a failure to provide synergistic support for the all important movement of the diaphragm. Remember, the diaphragm is the major muscle of breathing, and restoring its function as much as possible is the major objective of chest physiotherapy. This is why this type of therapy is generally known as "Diaphragm Breathing Exercises" or "Diaphragmatic Breathing Training" or just Diaphragmatic Breathing."

To provide the crucial synergistic support from the lower rib cage structures for optimal diaphragm movement the "Bucket Handle" movement must be restored. To achieve this, the focus of your inspiratory breath must be on the lowest-lateral ribs, at a point directly below the anterior portion of your arm pits. Placing your hands, or having an assistant place their hands in the correct position (as shown in the diagrams in the last section) is helpful in getting started. You should try to feel these lower ribs moving outward, and also try to feel air moving into this region. After some initial practice you should be able to perform this chest movement naturally, and without needing to have hand placement to remind you. And as your ribs become more mobile with Belt Exercises you will find this easier to do. And with improved rib excursions and improved chest mobility a larger and more satisfying breath is now possible.

And what about teaching specific diaphragm movement? Well, this is what you are doing by learning correct Bucket Handle rib movement. Remember, the diaphragm and the rib cage muscles performing the Bucket Handle movement act as a synergistic muscle group. By activating the Bucket Handle movement, the diaphragm movement will naturally follow.

Note the synergistic progressive flow of muscular group movement here. First is the rib cage Bucket Handle movement, and then soon after the upper abdomen begins to rise due to diaphragm movement into the abdomen. At this juncture, do not attempt to puff your upper belly out to aid inspiration. Continue to focus on the Bucket Handle movement, and the abdomen will rise on it's own with further diaphragm movement.

UPPER  CHEST  MOVEMENT

And what about reducing the abnormal upper chest movement? Almost always that movement will gradually go away if you simply maintain focus on the lower rib, Bucket Handle movement. I do not advocate, and in fact discourage the popular "Two Hand Technique," with one hand on the upper chest (to encourage minimal movement) and the other hand on the central upper abdomen (to encourage maximal diaphragm movement). As we have seen earlier, this technique unfortunately tends to teach the abnormal trick movement of Belly Puffing.

However, with very large breaths you will note the upper chest now moving upward. This is normal, as you are now activating the so-called "Accessory Breathing Muscles" driving the "Pump Handle" movement. This is a normal emergency breathing movement to provide maximal breathing. It can be easily seen as the "heaving" upper chest of an athlete who has just finished an exhausting race. Think of this movement as an emergency breathing reserve, to be encouraged. However, it is important to maintain focus on the lower Bucket Handle movement as is the dominant movement. Let the upper chest movement flow from the lower Bucket Handle movement.

Note the synergistic flow of muscle group activity. First the lower chest Bucket Handle movement, then the abdomen rises with diaphragm activity. Then, with larger breaths there is more Bucket Handle movement and more diaphragm activity and a further rise of the abdomen, and the upper chest now starts to rise with Pump Handle movement with very deep breathing.

UPPER  CHEST  MOVEMENT WITH ANXIETY

The "Pump Handle" upper chest movement is basically a defensive, emergency type of breathing. This is probably how it became ingrained as part of the abnormal "Unit Movement" of the "Barrel Chest" deformity.

However, it is very interesting to note, that anxiety will frequently trigger this type of upper chest movement, and it does so both in people with with COPD as well as those with perfectly normal lungs. Presumably this is because tension and anxiety is part of the overall defensive, emergency reaction.

In distressed patients with COPD having an acute dyspnea attack it may be impossible to tell if upper chest movement is at least partially due to this anxiety based type of breathing. Most likely most such upper chest movement in this situation is a part of a desirable muscular recruitment to assist breathing (i.e. due to deranged pulmonary mechanics resulting from Dynamic Hyperinflation). To resolve this problem it is best to focus on the lower chest "Bucket Handle" movement, and if in doubt about residual upper chest movement, try to voluntarily limit the upper chest movement.

Some people will immediately display upper chest breathing when starting the Rescue Breathing Pattern. These people should immediately try to limit upper chest breathing, while at the same time calming themselves.

In people with normal lungs suffering an anxiety attack or "Panic Attack" and an overbreathing condition known as the "Hyperventilation Syndrome" will frequently exhibit a heaving upper chest manner of breathing. The link between acute anxiety and this type of breathing seems so compelling, that many therapists make elimination of upper chest movement a priority in reducing anxiety and establishing breathing control.

EXPIRATION CONTROL

And what about expiration? Expiration is mainly about timing of the length of breathing out. Generally speaking, expiration should be entirely relaxed and passive, to allow sufficient time for the air to get out, and also permit rest of the respiratory muscles. However, if you do need to provide some muscular force to exhale, it is best done by gently tightening the upper abdominal muscles, and from there there will flow some exhalation activity to the lower ribs. Remember, if you need to forcefully exhale, do it as gently as possible, in order to minimize any "Dynamic Bronchial Compression," which will make the airways smaller, and therefore impair air flow. More on this subject later.

RESPIRATORY  MUSCLE  STRENGTHENING


The rib muscles of breathing, having been encased in the stiffened Barrel Chest deformity, have undergone at least some measure of atrophy and weakness. And unfortunately, when liberated from the stiff Barrel Chest they initially may be so weak they tire very easily. Most patients will gradually improve this muscle strength with increased activity made possible by breathing control, and progress well with their rehabilitation process. However, occasionally some initial post chest mobilization muscular strengthening is needed to speed the rehabilitation process. If so, a modification of the Breathing Belt technique can be used for this purpose.

To strengthen the rib muscles, perform the Belt Exercise as noted above. However, instead of suddenly releasing the belt and allowing the chest wall to spring out, gradually release the belt tension as you inhale, and force the expanding chest to work a bit. This requires a little practice to do properly, as maintaining a steady pressure as the chest moves out on inspiration is a subtle skill. Initially the pressure applied should be gentle. And as your strength improves, increase the inspiration belt tension until you are using a firm tension and working fairly hard to breathe in. These strengthening exercises should be done only two or three times a day, and never more than five minutes at a time. The amount of fatigue you feel at the end of this exercise will be your indicator as to whether or not you should increase of decrease the amount of belt tension.

Another useful option are the commercial "Inspiratory Muscle Training (IMT)" devices. These simple devices are quite economical. Your doctor will need to prescribe one for you, and if so, be sure to get a so-called "threshold" training device. The inspiration pressure needs to be set, and start with about 10 to 15 cm water pressure, and gradually work up to between 30 and preferably 40 cm water pressure. Some advocate using these devices for 15, 20 or even 30 minutes 3 or 4 times a day. I believe these are excessively prolonged, unpleasant, and unnecessary exercise sessions. Again, as with belt exercises I would suggest that your exercise sessions with these IMT devices be limited to five minutes, and only two or at most three times a day.

The  RESPIRATORY  SQUEEZE


The Respiratory Squeeze is basically an exaggerated Breathing Belt exercise. The object is to squeeze as much air out of your lungs as possible, in preparation for a better inspiration breath. The method can be used for rapid lung decompression of an overinflated lung causing an acute attack of dyspnea. And it may also be used to advantage in clearing retained bronchial mucus (phlegm) as part of the "Huff Cough" technique. More on this aspect in another module.

The Respiratory Squeeze is performed in the sitting position, with the knees touching. The Breathing Belt technique is then done as described above, but with a longer time spent on expiration, at least four or five or more times longer than your usual exhalation time.

However, instead of maintaining an upright posture, lean your body forward on expiration as you are applying belt pressure. As you near the end of expiration your hands should now be together in the center of your upper abdomen, and by leaning against your legs, your hands will assist in pushing your diaphragm upward for enhanced lung emptying. Then, on inspiration release the belt pressure and simultaneously return your body to the upright position, and feel the air rush in.

For correction of even severe lung overinflation, properly done, only one or two Respiratory Squeezes should be needed. For clearing stubborn sticky retained phlegm, repeating a Respiratory Squeeze before each Huff Cough maneuver can be very helpful.

Your chest should now be mechanically ready to learn how to breathe. We will now move on to bad breathing patterns, and why and how to correct them.

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Breathing  Control



BREATHING CONTROL
and
AlVEOLAR VENTILATION EFFICIENCY

When patients understand the underlying reasons why they have problems, and what they must do to correct these problems, they then become informed partners in their health care. Knowledgeable patients generally do much better than uninformed patients. For this reason it is important you have some understanding of breathing control, before we teach you how to alter your breathing patterns.

Breathing control is regulated by many sensors in the body that feed information into the Respiratory Center in the brain, which in turn sends out feedback regulatory signals to the diaphragm and rib muscles to tell them how to move to make a Breathing Pattern, in order for you to breathe in and out.

The system is very complex, with mechanical sensors in the lung and chest wall that sense pressure and tissue stretch, and the volume position of the lung. There are also chemical sensors in the central blood vessels and within the brain that sense Oxygen and Carbon Dioxide levels in the blood. The Respiratory Center must make a constant and delicate balance of breathing, to be certain enough Oxygen in breathed into the body, and that the waste Carbon Dioxide from body metabolism is removed. This system is very dynamically active, and constantly changing, to adapt to changing metabolic needs, such as when you go from a resting to exercise condition.

The respiratory regulatory system is unique among all the major body systems, because it has given you the ability to voluntarily control your breathing patterns. Higher centers in the brain can easily override the normal automatic feedback regulatory system of the Respiratory Center. Think of this as a manual override button, which allows you to take larger or smaller breaths, or vary your breathing rate and pattern in subtle ways. This ability to voluntarily control your breathing, and to train your breathing into a new pattern, is the marvelous tool that you can use to correct your abnormal COPD Breathing Pattern, and thereby minimize your breathing distress.

There is however a downside to this ability to voluntarily alter your breathing pattern. As noted in the discussion on the Rescue Breathing Pattern, this psychologically driven rapid and forceful breathing pattern can have serious consequences. This is particularly true in the COPD patient who is in danger of promoting Dynamic Hyperinflation and making their breathing distress much worse.

The COPD / Emphysema Breathing Pattern


The person with COPD typically breathes at a relatively rapid rate and with a small breath volume, and usually with a relatively short expiration time. This is due to abnormal mechanical factors within your lungs and chest wall. The lungs are frequently stretched-out near their elastic limit and therefore more effort has to be expended to make the lungs move. The chest wall and chest muscles of breathing are also stretched-out near their elastic limit, and furthermore are involved in the stiffening problem related to the Barrel Chest deformity, thus making the chest wall even harder to move.

The result is a small breath volume (called the Tidal Volume), because taking in a larger breath is just too hard to do, and would require too much so-called Work of Breathing. Shortness of breath (Dyspnea) is related to a number of factors, but is most closely related to the Work of Breathing. And because of these smaller breaths, in order to provide enough air, the respiratory rate of breathing must speed up, hence the COPD breathing pattern of rapid and shallow breathing.

Unfortunately however, using smaller breaths to breathe more easily has a serious downside. Not all of the air you breathe in actually does you any good. Only the air that reaches the alveoli (the air sacks) can participate in the Gas Exchange of Oxygen and Carbon Dioxide with the blood. This air, at the beginning of the breath, that penetrates down deepest to the alveoli, is called the Alveolar Ventilation. The air at the end of a breath in does not penetrate the lungs deep enough to reach the alveoli and participate in Gas Exchange, and appropriately is called Dead Space Ventilation. The smaller your breath, the smaller will be your relative Alveolar Ventilation that is actually doing you some good, by permitting gas exchange of Oxygen and Carbon Dioxide.

Yes indeed, smaller breaths are easier to do, but they carry this serious breathing penalty of having relatively more of your breath as unused Dead Space Ventilation. Being able to take a larger breath with relative ease is crucial to optimizing your breathing. You now know why we have spent so much time discussing the chest wall, and the mobilization and strengthening of the chest wall, in order that you may take in a larger breath of air, and doing it more efficiently and easily.

This also introduces you to a more complex and comprehensive concept of balancing, and trading-off various parts of your breathing, in order to best optimize your breathing pattern within the constraints imposed by the disordered mechanics of the COPD and Emphysema lung. Later you will learn more about other apparent contradictions that may be used in a balanced manner to optimize your breathing, and how the Breathing Trainer may help you in fine tuning these balancing factors to advantage.

But first you must understand the concept of lung overinflation, and in particular the all-important concept of Dynamic Hyperinflation, as this is substantially responsible for increased breathing distress, literally having been tricked into this situation by an abnormal breathing pattern. This will be discussed in the next module.

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Dynamic Hyperinflation


DYNAMIC  HYPERINFLATION

Note: In order to easily follow the description of this diagram, it is suggested that you first print the diagrams.
You can do this by right clicking on the diagram, and then use the commands "View as" or "Copy" (or similar words).
This will put the diagram in the computer clipboard, and you can print it from there.

Lung Overinflation or Hyperinflation is a critically important component part of both COPD and Emphysema, and particularly so with Emphysema. In fact, recent evidence suggests that correction of Hyperinflation appears to be more important in the relief of dyspnea, than is the correction of airway obstruction with broncodilator therapy. Said another way, if you use so-called Rescue Drugs such as Metered Dose Inhalers to relieve dyspnea caused by airway bronchospasm, the majority of your dyspnea relief comes not from the bronchospasm relief, but rather as a result of relieved bronchospasm now permitting correction of hyperinflation.

This section will tell you how to obtain further dyspnea relief, after you have used your bronchodilator medications. Bronchodilator medications are very important, but they are only the first step to obtain maximal dyspnea relief.

There are two general types of overinflation. The first is so-called "Anatomic Hyperinflation" seen in Emphysema, where there is actual destruction of alveolar lung tissue, to create enlarged cystic overdistended air spaces.

The second general type of overinflation is so-called "Physiologic Hyperinflation" seen in both COPD and Emphysema. The underlying problem here is the airway obstruction common to both conditions. With increased airway obstruction causing increased resistance to air flow, the lung may not have enough time to empty before the next inhaled breath.

Remember, on breathing in, all the structures in the lung, including the airways, get larger, and therefore air moves into the lung relatively easier on inspiration. Conversely, on breathing out, everything in the lungs, including the airways, gets smaller. Therefore, it is always relatively more difficult to to get air out of the lung on expiration. As a result, some air is trapped in the lung, causing it to overinflate.

This diagram is from 1955 first edition of The  Lung, by Dr. Julius Comroe et al.
The lung is depicted as a single air sack, and the arrow indicates air moving in and out of the lung. The dark wavy line below is the subject breathing in and out.
Figure A shows the normal condition, with air moving out freely, and no lung overinflation.

Figure B shows some airway obstruction, and therefore "Air Trapping" on expiration, and with overinflation developing. Note the breathing tracing moving upward.

Figure C shows even more airway obstruction, and the resulting increased overinflation.

The problem here is not enough time for the lung to empty on expiration. The so-called Time Constant required for lung emptying has been exceeded. Note carefully, the faster you breathe, the worse this problem will become.

The older term "Physiologic Hyperinflation" is now evolving into the name "Dynamic Hyperinflation," and more recently has generally been used as a phenomenon related to patient exertion. However, this is not entirely correct, as it is now clear that this type of hyperinflation is commonly present to some degree, even with mild to moderate airway obstructive disease, even while patients are at rest. Dynamic Hyperinflation therefore is of two general types, "Resting Dynamic Hyperinflation" and "Active Dynamic Hyperinflation."

The importance of this surprising recent observation that Dynamic Hyperinflation is frequently present at rest in mild to moderate airway obstructive disease is not that it is causing significant dyspnea at rest. In fact, it generally is not of significance. However, it is clear that these generally asymptomatic patients are indeed vulnerable to further exacerbation of their Dynamic Hyperinflation should they increase their breathing rate for exertion or whatever reason, and therefore have an exaggerated dyspnea response. Clearly, this problem can no longer be considered as a significant factor only in severe COPD.

ACTIVE  DYNAMIC  HYPERINFLATION

      Active Dynamic Hyperinflation in COPD occurs most commonly:
    * With general increased effort and Exertion
    * After an uncontrolled Coughing spell
    * With the Rescue Breathing Pattern

The Rescue Breathing Pattern


The Rescue Breathing Pattern ("RBP") may be briefly characterized as "....trying to pump air in and out of your lungs as fast and as hard as you can...."

It is a basic psychological cognitive reflex (i.e. controlled by a persons thoughts), generated by a persons conscious will to try and breathe in a manner to relieve acute dyspnea distress. It is not a part of the complex traditional mechanical feedback reflexes from the lung, or the blood chemical (Oxygen and Carbon Dioxide) feedback mechanisms, that automatically control breathing via the Respiratory Center in the brain. This cognitive reaction to dyspnea distress is seen commonly, in both people with normal lungs, and those with COPD diseased lungs. It is perfectly normal and natural for patients with COPD to get upset and anxious if they are experiencing increased shortness of breath, and to react with the Rescue Breathing Pattern. Unfortunately this reaction will only make their dyspnea worse.

If you have COPD and have an acute breathing attack, the more you struggle to catch your breath by breathing rapidly, the worse your problem will become. This is because of a so-called "Vicious Circle" phenomenon, because the faster you breathe, the less time you have to get air out of your lungs. This is physiologic disaster, because it makes Dynamic Hyperinflation progressively worse. It is also a sad and paradoxical reality, that your natural instincts to help yourself, should in fact be turned against you, to make your breathing attack worse.

Rapid breathing, for whatever reason, will trigger this vicious circle response, and produce Dynamic Hyperinflation to make your breathing worse. Literally, and in fact, the basic mechanics of COPD breathing have been tricked into what can only be considered as a self destructive abnormal breathing response. And this is why learning recognize and control the emotional aspects of the Rescue Breathing Pattern, and why breathing control in this situation is so important, because your natural breathing defense mechanisms have been turned against you.

It is very important you clearly understand, if you have a mild episode of increased dyspnea, and then become anxious and upset, you may trigger the Rescue Breathing Pattern and rapidly make your dyspnea attack much worse. This is because the increased breathing rate of the RBP produces Dynamic Hyperinflation. Remaining calm and not allowing yourself to become upset by your dyspnea is a critically important component of COPD breathing control to prevent or minimize acute dyspnea events.

Typically in this situation, patients use their so-called Rescue Medications, usually a Metered Dose Inhaler, to relieve acute dyspnea exacerbations. This is desirable treatment, but rescue drugs are only the beginning of the process to obtain full dyspnea relief. This is because, even after complete, 100% maximal bronchospasm correction achieved by medications, the patient is still left with their original underlying problem of severe airway obstructive disease. It should therefore be obvious that it is imperative that you learn breathing control to prevent and/or fully correct this problem of Dynamic Hyperinflation.

In other modules we will show you how to control Dynamic Hyperinflation with exertion, and after uncontrolled coughing spells. These future lessons will all be based on what you have learned here with the Rescue Breathing Pattern response.

RESTING  DYNAMIC  HYPERINFLATION


A recent large bronchodilator study involving some 957 patients revealed that 48% of these patients had Resting Dynamic Hyperinflation. Clearly, Resting Dynamic Hyperinflation is a major problem in the COPD population.

What is not clear is, how many patients among those 48% had their Resting Dynamic Hyperinflation fully resolved by their bronchodilator therapy. Until this question is answered, it would seem prudent that all patients with symptomatic COPD have breathing control skills to determine whether or not these skills can enhance their overall resting breathing comfort.

This study makes it very clear, that almost half of the COPD population, even while stable and at rest, are critically vulnerable to any increase in their breathing rate, and that any increase in their breathing rate may precipitate them into acute Active Dynamic Hyperinflation.

Well, since the emptying of the lung on expiration is generally by passive elastic recoil of the chest wall and lung, why not solve the problem of hyperinflation by simply exerting voluntary muscular force to expiration and force the air out? Sadly, the answer to that question varies somewhere between "Yes" and "No." To understand this problem we will explain the subtle and important problem of "Dynamic Bronchial Compression" in the next module.

As to all Dynamic Hyperinflation therapy, always remember, it is mainly an expiration TIME problem. You must learn to manipulate your breathing pattern to generate enough time to allow appropriate emptying of your lung on exhalation. We will show you later how to adjust your breathing time constraints with the Breathing Trainer, to balance the conflicting constraints within a breathing pattern, and give you enough time to breathe out in an optimal manner.

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Dynamic Bronchial Compression


DYNAMIC BRONCHIAL COMPRESSION

The lung has no muscles (other than the muscles surrounding the larger bronchial airways) to cause it to expand and contract, to pump air in and out. The lung in fact is a totally passive elastic structure that for inspiration depends on the chest wall and diaphragm to literally suck it outwards, and therefore expand the lung, to pull air into the alveoli. This is essentially what we have been discussing under diaphragmatic breathing.

The act of expiration under normal conditions is passive, and simply involves the chest wall and diaphragm relaxing, and the elastic forces within the the lung and chest wall that have been stretched out by inspiration, now retract to their resting state. The lung therefore collapses and pushes the stale air out.

There are muscles in the rib cage and that can actively cause the rib cage to contract, and push air out. And if the muscles of the abdominal wall contract, they cause the diaphragm to be pushed up into the chest cavity, and press on the lungs, and thereby further assist active expiration. Normally however these expiratory muscles are not used, except during conditions of exercise, where they are used to literally pump air in and out of the lungs.

So, if Dynamic Hyperinflation correction needs to get air out of the chest, why not use these expiratory muscles to actively push that stale air out? The answer is, yes, you can do this to assist in expiration, but there is a special problem here for the COPD and Emphysema patient. And this problem is called Dynamic Bronchial Compression.


This diagram simplifies the lung down to one alveolus and one bronchial tube within the chest wall, and the bronchial tube leading to the outside air.
When the lung exhales, the chest wall retracts and moves in and therefore applies pressure and everything within the chest gets smaller. The pressure applied to the alveoli is desirable, because that is what pushes the stale air out.

However, this same pressure is also applied to the bronchi, and also makes them smaller, and that is not desirable, because the bronchial tubes also become narrower, and therefore impose a greater degree of airway obstruction for the stale air trying to get out. In the normal lung this is not a problem, but in COPD, and particularly with Emphysema, the bronchial walls are diseased and narrowed, and they are less well outward supported by diseased and deficient elastic structures. These COPD bronchial tubes therefore are much more susceptible to collapse, and they collapse prematurely at particularly weak areas, when the so-called Critical Closing Pressure of these airways is exceeded. The expiration collapse of a regional area of the smaller bronchial tubes is depicted in the diagram.

If you now apply active muscular pressure on expiration, the internal chest pressure will be higher than normal, and the problem of Dynamic Bronchial Compression will be exacerbated. Therefore the airways will collapse prematurely, and to a greater extent, and the problem of getting stale air out of your chest will be made worse. And furthermore, forced exhalation increases the expiratory Work of Breathing, and can be very exhausting.

Here again you have an example of the problem of conflicting actions within the physiology of breathing, and the need to balance these conflicting forces, namely, desirable passive expiration versus active expiration muscular contraction to help get overinflation stale air out of your lungs.

Forced active expiration, and particularly chronic forced expiration, is seldom used as a routine technique, because it is usually exhausting.

However, as a "Rescue Technique," to help correct Dynamic Hyperinflation, gentle forced expiration, applied in a controlled manner, can be very helpful. And the occasional patient with particular problems of emptying their lungs properly, can use a modification of this technique in a chronic manner.

This technique should not be done throughout all of expiration. As with all expiration, the initial act of breathing out should be done in a totally relaxed manner, and this relaxation should be continued as long as possible. However, when it is apparent the lung is not going to empty within a reasonable time, the Rescue Technique of forced expiration should be applied. This is usually at approximately two thirds to three quarters of the way through expiration. The exhalation muscular force must be very deliberately and gently applied.

Think of this force as though you are wringing water out of a wet bath towel. If you apply a strong,sudden force, you will get out a certain amount of water. If however you apply a gentle firm squeezing force, over a longer period of time, you will get out more water, and with less overall effort. Learn to apply this expiration squeezing force within your chest as gently as possible, and with just enough force to get the air out within an appropriate amount of time, that is, the end of expiration. Doing so will minimize the problem of aggravating the Dynamic Bronchial Compression problem, and will make overall expiration easier.

With the Breathing Trainer it is easy to see not only how long you should be breathing out, but also where you should apply this controlled expiration force.

So there you have it, the desired major component parts of the therapeutic COPD breathing pattern, and all of them conflicting with one another to a greater or lesser degree:

    * Breath size (Tidal Volume)   (LARGER)
    * Respiratory Rate    (SLOWER)
    * Expiration Time   (LONGER)
    * Expiration    (PASSIVE)

All of these component parts must be balanced carefully, to get an optimal breathing pattern for your individual needs.

So far you have spent a lot of time learning about the underlying complexities of breathing training. If you understand why these various breathing recommendations are made, you will be better able to utilize, and to work with these recommendations, and to fine tune them to your individual needs for increased comfort.

The more you know, the better you will do in chest physiotherapy and breathing training.

We will now show you some COPD breathing patterns, and what to do about them. 

We have run out of space in this Knol.

Go to Part 2 of this series

This series, including some video material, may also be viewed on my web site at:
http://www.sierrabiotech.com/bt_copd_home.html

Comments

low dose naltrexone for copd.

i would like to add that low dose naltrexone works very good for copd patients.my friend has much more energy,uses the inhaler much less and call it a wonder drug.there is no side effects.

you can find more info here.

http://www.lowdosenaltrexone.org/

Last edited Aug 6, 2008 8:18 PM
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Deane Hillsman
Deane Hillsman
Pulmonary Physician
Sacramento, California
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